Vitamin C and Belly Fat

Winter weather seems to magnify all your aches and pains, especially if arthritis is involved.

But there’s a vitamin that can go a long way in soothing those pains…

Recently I wrote about how squatting more, and taking vitamin C, can help your joints produce more synovial fluid — your body’s natural lubricant.

But another way to ensure joint health is to maintain a healthy weight…

Research shows that losing just ten pounds over ten years may cut your risk of developing arthritis by more than 50 percent!

Of course, losing weight can’t repair damage that’s already been done to your joints, but it can help decrease pain and slow further progression of arthritis.

And you may be surprised to learn how much vitamin C can help here, too…

That dangerous ‘spare tire’ cycle

In addition to causing pain in arthritic joints, carrying too much belly fat puts you at risk for serious life-threatening consequences.

Excess abdominal fat not only releases an overabundance of hormones that alter cholesterol levels, raise blood pressure and reduce insulin use — but these poorly functioning hormones also make is easier for you to pack on the weight.

This puts you at higher risk for heart disease and stroke — and this metabolic nightmare creates a cycle that makes it harder for your body’s systems to work together to keep you healthy.

So, how can vitamin C help?

Your secret weapon for weight loss

Vitamin C and glucose have similar chemical structures. They are constantly competing to get into your cells, and vitamin C is at a distinct disadvantage.

There is a receptor called the Glut-1 receptor that allows both glucose and C to enter our cells. Unfortunately, it likes glucose a lot better! This means that vitamin C has to show up in greater numbers in order to get in.

When you are vitamin C deficient, much more glucose enters your cells than what your body requires to meet its daily energy needs. This triggers the production of insulin.

Insulin helps the body absorb glucose, while at the same time telling your body to stop burning stored fat and to turn its attention to absorbing what’s coming in.

See the connection? Not enough vitamin C … too much glucose … more insulin … less fat-burning …. spare tire.

For exercise to work, you need your C!

Experts at Harvard University Medical School recommend moderate physical activity at least 30 minutes per day as optimal for reducing belly fat. Now, if you have arthritis pain you may be limited as to how much you can do — so this is why ample vitamin C is especially beneficial to you…

Without adequate vitamin C, your body will not burn fat as efficiently as it should, even with a good exercise regimen.

Quite simply, vitamin C is needed for fat metabolism. So, without enough of it, you won’t lose much belly fat, even if you exercise!

Related: 5 ways cinnamon keeps you thin and healthy

One study found that people with adequate intake of vitamin C oxidize 30% more fat during moderate exercise than those with a low intake.

Vitamin C prevents adrenal ‘panic’ and weight gain

Supporting your adrenal glands with adequate amounts of vitamin C can short-circuit another chain of events that results in weight gain.

The highest concentration of vitamin C in the body is found in the adrenal glands. vitamin C is essential to produce cortisol, the ‘fight-or-flight’ stress hormone.

Without an adequate supply of vitamin C, your adrenal glands ‘panic,’ and actually produce more cortisol. This wreaks havoc on your blood sugar which, as we have seen, leads directly to the accumulation of that unhealthy belly fat.

Making vitamin C part of your daily diet

Be careful about looking to juices to get your vitamin C, because you’ll also get a boatload of added sugars. Vitamin C is readily available in a wide range of fruits and vegetables. Some of those with the highest concentration of C are:

• Sweet red pepper, ½ cup raw, 95 mg.
• Kiwi fruit, 1 fruit, 91 mg.
• Strawberries, 1 cup, 85 mg.
• Orange, 1 medium, 70 mg.
• Broccoli, ½ cup cooked, 51 mg.
• Grapefruit, ½ medium, 38 mg.
• Tomato, 1 medium, 16 mg.
• Spinach, 1 cup raw, 8 mg.

Related: What strawberries do for your body

You can also take a daily vitamin C supplement. The daily recommended intake is at least 65-75 mg. per day to avoid deficiency, but that’s the bare minimum. A higher dosage of 200 to 400 mg. per day has been shown to bring vitamin C levels up to a steady state in the blood.

However, any amount beyond 200 mg. at a time is largely excreted. So, to get the maximum absorption and benefit, it’s best to take no more than 200 mg. per dose.

Editor’s note: In his book, The Insulin Factor, Dr. Michael Cutler reveals compelling facts on how your master hormone, insulin, plays such a critical role in the health of your entire body. It’s a must-read, and the only book you’ll find that reveals the insulin link to cancer and heart disease, and how you can stop this from happening to your body — despite the best efforts of Big Pharma. Your doctor probably won’t tell you about this, so check it out yourself by clicking here.

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Vitamin C

- Vitamin C, Lysine, Proline + Some Antioxidants

To normalize your cholesterol and/or high blood pressure, while gently healing your arteries and removing plaque:

RECOMMENDED ADULT DAILY DOSES OF VITAMIN C, LYSINE AND PROLINE
Seriousness of Condition	Vitamin C	Lysine	Proline
Prophylactic/Prevention	3 g	3 g	0.5 g
High risk Therapeutic (mother /father /sister /brother hashad a heart attack or stroke)	6 g	6 g	1.0 g
Serious Therapeutic (for someone who has had a heart attack or stroke)	9 g	9 g	1.5 g

For information onAntioxidants and other useful supplements to aid the reversal of atherosclerosis and maintain health:

Tools against CVD

For instructions on preparing and taking the Rath/Pauling Therapy ingredients:

How to do the Rath/Pauling Therapy

Effects of the Rath/Pauling Therapy in CVD

Addresses the root cause of atherosclerosis in CVD by strengthening and healing blood vessels

–   Amino acids proline and lysine are basic building blocks of collagen - additionally, lysine promotes pituitary gland secretion of hGH (human growth hormone), the master hormone that will indirectly promote fibroblasts to produce more collagen throughout the body.

Vitamin C is involved in collagen production;

–    Inhibits the binding of Lp(a) molecules to the blood vessel walls – thus removing plaque build-ups.

–   Sufficient lysine and proline work to unbind Lp(a) from the arterial wall - by blocking the collagen-digesting enzyme anchor sites in the connective tissue.

Lowers Lp(a) blood levels over time and keeps them low

Therapy mechanism doesn't depend on the cause of plaque formation

It doesn't matterif the arterial lesions were caused by mechanical stress, a vitamin deficiency, oxidized cholesterol, elevated homocysteine or sugars, fat in the diet, or even . . .

Little green men!

Rath/Pauling Patents

According to the Rath/Pauling 1994 U.S. patent, a binding inhibitor (E.g. lysine or lysine analogs) together with vitamin C can stop and even reverse plaque formations - the amino acid lysine (lysine analogs), along with vitamin C and other antioxidants (E.g. Co-Q10, vitamin E and vitamin A) in sufficient concentration, can inhibit Lp(a) from binding to exposed lysine residues.

US Patent No. 5,278,189, Pauling/Rath (1994).Prevention and treatment of occlusive cardiovascular disease with ascorbate and substances that inhibit the binding of lipoprotein (A)

Another patent concerns stripping plaque from transplant organs(actually filed first) -in later experiments Pauling and Rath showed that proline residues are also exposed by lesions in blood vessels; they filed a U.S. patent for using proline as well as lysine, vitamin C, other amino acids and antioxidants (in oral amounts well past what is needed for prevention) to inhibit Lp(a) binding and so “melt away”atherosclerotic plaques in human organs dipped in these solvents during organ transplants

US Patent No. 5230996: Pauling/Rath (1993). A Procedure for the Cleansing/Removal of Atherosclerotic Plaque from Human Organs During Transplant Surgery.

Reported successes

Orthomolecular physicians have reported much success in treating heart disease with synergistic combinations of ascorbate, lysine and proline

Here's just a couple of testimonies:

–   Linus Pauling Case History: a National Science Medalist who had undergone several coronary artery bypass grafts (CABGs), each of which had re-clogged, and who had been prescribed statin drugs for high cholesterol as well as calcium channel blockers and beta-blockers for high blood pressure. After discussing his history with Pauling, this patient began a supplement program, including 6g of vitamin C; however, his condition continued to worsen. Pauling then suggested adding L-lysine (peaking at 6g/day) to his cocktail. The patient described the results as bordering on miraculous: his walking distance suddenly recovered, and he was again able to do his own yard work (including the cutting up of a tree with his chainsaw and the painting of his house).

–   Dr. Kathie Dalessandri, MD: reported her own dramatic improvement in the Archives of Internal Medicine after using vitamin C and lysine. “I am a 53-year-old woman with a significantly elevated level of Lp(a) (27 mg/dL). . . I began to follow the advice of Linus Pauling. For individuals who have an Lp(a) level higher than 25 mg/dL and a family history of heart disease, the recommendation is to take 3 g/d of both ascorbic acid and L-lysine monohydrochloride.After 6 months of this regimen, with no adverse effects, my Lp(a) level decreased to 14 mg/dL, a reduction of 48%.”

Studies connecting Vitamin C and CVD

1950's - Canadian physician, Dr. G. C. Willis demonstrated that an ascorbate deficiency increased cholesterol synthesis in animals

Increased dietary cholesterol reduced vitamin C levels - and, conversely, vitamin C supplementation decreased cholesterol levels.

Willis found that Vitamin C reversed atherosclerosis in guinea pigs -like humans, they do not produce their own ascorbate

Willis GC. 1953. An Experimental Study of the Intimal Hemorrhages and in the Precipitation of Coronary Thrombi. Canadian Medical Association Journal, vol.69:pp.17-22.

Willis et al GC. 1954. Serial Arteriography in Atherosclerosis. Canadian Medical Association Journal, vol.71: pp.562-568.

Willis GC, Fishman S. 1955. Ascorbic Acid Content of Human Arterial Tissue. Canadian Medical Association Journal, vol.72:pp.500-503.

Willis GC. 1957. The Influence of Ascorbic Acid upon the Liver. Canadian Medical Association Journal, vol.76:pp.1044-1048.

Willis GC. 1957. The Reversibility of Atherosclerosis. Canadian Medical Association Journal of Nutrition, vol.77:pp.106-109.

1971 - British physician, Dr. Constance Spittle, demonstrated that vitamin C therapy could lower or raise cholesterol depending on CVD presence

–    Patients exhibited a transitory rise in blood cholesterol when given vitamin C therapy –explained by cholesterol released from plaques as vitamin Chealed the blood vessel walls;

–    Patients with no CVD showed lower blood cholesterol levels.

Spittle CR. 1971. Atherosclerosis and Vitamin C, The Lancet, Dec 11;(18):pp.1280-1.

Vitamin C

Read read read read!

Vitamin C

Natural eradication of high cholesterol

How Vitamins and Other Nutritional Supplements Can Help Patients With Elevated Cholesterol Levels

Interestingly, some patients report a transitory rise in cholesterol levels when they start taking vitamins. Because the rise in blood cholesterol levels is not the result of increased cholesterol production, it has to come from other sources — primarily atherosclerotic deposits in the artery walls. This important mechanism was first described by Dr. Constance Spittle in the medical journal The Lancet in 1972. She reported that vitamin supplementation in patients with existing cardiovascular disease frequently led to a temporary increase of cholesterol levels in the blood. In contrast, the cholesterol levels of healthy test persons did not rise with vitamin supplementation.

The temporary rise in cholesterol is an additional sign of the healing process in the artery walls and the decreasing of fatty deposits. The mechanism described here is, of course, not only valid for cholesterol, but also for triglycerides, LDL, lipoprotein (a) and other secondary risk factors, which have accumulated over decades inside the artery walls and have been slowly released into the bloodstream.

Should your cholesterol levels rise when you start following these recommendations, it can indicate the reversal of existing deposits in your artery walls. You should continue the vitamin program until, after several months, the blood level of cholesterol decreases below the initial values. A diet high in soluble fiber (e.g. oat bran, cereals and pectins) can further decrease cholesterol and other secondary risk factors in the blood.

The effect of vitamin C on the blood levels of cholesterol and other blood fats has been documented in numerous clinical studies. More than 40 of these studies have been evaluated by Dr. Harrie Hemilä of the University of Helsinki, Finland. In patients with high initial cholesterol values (above 270 mg per deciliter), vitamin C supplementation was able to decrease cholesterol levels up to 20%. In contrast, patients with low and medium initial values of cholesterol showed only a slight cholesterol- lowering effect or the levels stayed the same.

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Health benefits of Vitamin C

Supplemental Vitamin C and CoQ10 are completely nontoxic and would lessen or eliminate most of these statin-induced effects.  More headlines from BOLENREPORT.COM illustrate the health benefits of Vitamin C: 

1.  HARVARD: VITAMIN C ONLY 1 OF 880 SUBSTANCES TO REGENERATE HEART MUSCLE FROM STEM CELLS

2.  15-YEAR HARVARD STUDY OF 85,000 FINDS SINGLE VITAMIN C PILL REDUCES HEART DISEASE ALMOST 30%

2. THE RISK OF STROKE WAS 70% HIGHER AMONG THOSE IN THE LOWEST QUARTILE FOR SERUM VITAMIN C THAN AMONG THOSE IN THE HIGHEST

3.  VITAMIN C INHIBITS LIPID OXIDATION IN HUMAN HDL

4.   MORE VITAMIN C AS PILLS REDUCE CATARACTS BY 77%

5.  HIGH-DOSE VITAMIN C COMPLETELY PREVENTED DRUG-INDUCED AMNESIA IN MICE

6.   CARNITINE, ITS BUILDING BLOCKS VITAMIN C AND LYSINE, INCREASE MUSCLE STRENGTH

7.  MATTHIAS RATH CLAIMS CANCER HALTED WITH VITAMIN C/LYSINE/PROLINE AND GREEN TEA EXTRACT

8.  VITAMIN C BOOSTS IMMUNE SYSTEM IN AS LITTLE AS 5 HOURS (NIH)

9.   VITAMIN C PILLS EXTEND LIFE 6-YEARS (USC)

10. VITAMIN C (AND LYSINE) HALT ATHEROSCLEROSIS

11. IV VITAMIN C REVERSES ENDOTHELIAL DYSFUNCION

12. VITAMIN C - NEW TREATMENT FOR OSTEOPOROSIS

13.  VITAMIN C CAN (1) PREVENT THE DETERIORATION COMPLETELY; AND (2) CURE EVEN LARGE AORTIC ANEURYSMS WITHOUT SURGERY.[*]

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Vitamin C beats Statins!

The solution to high cholesterol and therefore heart attacks - tell us Astra Zeneca and Pfizer, two of the heavyweights in pharmaceutical remedies - is to take their drugs, Crestor and Liptor respectively.

According to what Dr. Mercola tells us in one of his recent articles - Crestor and Other Statins: Are They Really Worth the Risk? - there are serious side effects to these drugs, one of them being that your muscles start to waste away. The side effects are many and potentially serious, so they must be outweighed by the positive effects - after all drugs are rigorously evaluated according to the principle of risk/benefit analysis, where the benefit supposedly always outweighs the risks.

Well - that's the theory. In practice, we find that on the benefit side, those heavy statins are supposed to "lower cholesterol", a goal that may or may not have anything to do with the frequency of heart attacks. Correct that to reading "they lower bad cholesterol" - the low density lipoproteins - but really, there is not much difference. Mercola in his article cites two recent studies showing that with or without the medication, frequency of heart attacks did not significantly change.

The real solutions

One of the alternatives to these obviously inefficient and dangerous drugs is to eat more fish, or to obtain the oils found in fish by popping a fish oil supplement. That is an approach based on experience - people living in the far north such as the Eskimos - experience much fewer heart attacks than we do and one obvious difference is that they eat much fish supplying the beneficial omega-3 fatty acids.

Another alternative is to see what Dr Rath has found out about heart attacks. His cellular medicine maintains that illnesses occur at the level of cells. It's not the organs that are sick, in other words, but the cells are malnourished or otherwise impeded in their normal tasks.

In his book Why Animals Don't Get Heart Attacks, But People Do! Rath explains that heart attacks are due to a simple deficiency in some basic nutritional elements, which can be easily corrected for pennies a day. Of course the snake oil salesmen of the pharmaceutical camp will tell you this is preposterous. But take a moment to reflect. Who has got billions of dollars of revenues at stake? Read Rath's book or if you want, go to Rath's research site and explore. You will find a host of clinical studies that support the view of vitamin C deficiency being strongly implicated in heart disease.

The solution is simple. Take one or more grams of vitamin C daily. Yes, that's grams, like in a thousand milligrams. The 60 to 90 milligrams our health authorities tell us are quite sufficient for all needs, they leave us virtually starving for the nutrient.

You can confirm this by finding out how much vitamin C any mammalian body (except human bodies and those of a handful of other species) produce daily. It is in the range of several grams a day, to be exact, Linus Pauling says the range is 10 to 12 grams in proportion to the size of a human body.

Since we do not produce, by virtue of some genetic change that happened a long time ago, any vitamin C ourselves, our diet is the only way to get the substance. Vitamin C happens to be a vital ingredient for the production of collagen, exactly the material that blood vessels are made of. When blood vessels degenerate, we have a repair mechanism kicking in - cholesterol - which forms plaque in the arteries to stop them from leaking. What our pharmaceutical medical paradigm tells us that cholesterol is high when people have heart attacks, therefore we have to take drugs to "lower the cholesterol". Never mind that the cholesterol is the repair crew sent in to fix up our leaky arteries. We do everything possible to down size the repair crew and of course heart attack statistics keep going up and up and up.

If we had simply eaten our fruit (lots of it) or taken our vitamin C, as Pauling recommends, we would have eliminated the cause of heart attacks - degrading arterial walls - and on the way we would also have lowered our cholesterol. Naturally, cheaply and definitely.

Now I suppose that's too simple for our health authorities.

Some facts about cholesterol and heart disease

* ...the Journal of the Pharmaceutical Society of Japan, it was stated that "high total cholesterol is not positively associated with high coronary heart disease mortality rates among general populations more than 40-50 years of age. More importantly, higher total cholesterol values are associated with lower cancer and all-cause mortality rates among these populations." [Yakugaku Zasshi 125:833-52, 2005]

* It's a documented fact that as many adults experience a mortal heart attack with a total cholesterol count under 200 as over 300. [Medical World News March 27, 1992]

* About half of the patients who are admitted to hospitals for a heart attack have normal cholesterol levels. [Atherosclerosis 149: 181-90, 2000]

* More than 500,000 adult Americans experience a sudden mortal heart attack and have low-to-normal cholesterol levels and no more than 60% narrowing of their coronary arteries, not enough to impair oxygen flow to the heart or brain.

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Vitamin C and Mortality

VITAMIN C AND MORTALITY

Vitamin C’s proven effect on mortality stands in stark contrast to the statin drugs. According to author Bill Sardi, “ Vitamin C is the only vitamin repeatedly proven to increase the human life span when taken in doses that exceed dietary levels of this vitamin."   This observation was recently confirmed in Britain, again.

Low Blood Vitamin C Concentrations in the Older British Population Strongly Predict Mortality

In November 2003 the British Centre for Ageing and Public Health, London School of Hygiene and Tropical Medicine, United Kingdom. (astrid.fletcher@lshtm.ac.uk) published their findings of a direct correlation between low vitamin C and increased mortality.

We found a strong inverse relationship for blood ascorbate (vitamin C) concentrations with all-cause and cardiovascular disease mortality, which were only marginally reduced after adjustment for confounders or supplement use. Those in the lowest fifth (< 17 micromol/L) had the highest mortality, whereas those in the highest fifth (> 66 micromol/L) had a mortality risk nearly half that (hazard ratio = 0.54; 95% CI: 0.34, 0.84). Similar results were found after the exclusion of those subjects with cardiovascular disease or cancer at baseline (hazard ratio = 0.51; 0.28, 0.93). In fully adjusted models, there was no evidence for an influence of alpha-tocopherol (vitamin-E), beta-carotene, or retinol (vitamin-A) on total mortality. Dietary antioxidants measured by the food-frequency questionnaire were not associated with all-cause or cardiovascular disease mortality”

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Vitamin C v Statins

What is most horrifying about this problem is that cholesterol balance can be achieved without drugs, simply and safely by taking 2000-3000 milligrams of vitamin C per day for an adult. Unfortunately, vitamin C was misclassified as a micronutrient in the 1930s and 1940s, rather than an essential nutrient involved in dozens of body processes. Our health authorities recommend that we take only 60 milligrams per day, barely enough to prevent scurvy.

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Vitamin C and Statins

Purpose: Atrial fibrillation (AF) is the most frequent arrhythmia that follows coronary artery bypass graft (CABG). Patients developing postoperative AF (POAF) have significantly higher mortality rates. The consistent prophylactic effectiveness of statins and vitamin C are well-accepted; however, no evaluation on combined therapy has been performed. We aimed at assessing the efficacy of combination therapy with statin and vitamin C in comparison with statin alone in the prevention of post CABG-AF.

Methods: In a randomized double blind clinical trial, 120 candidates of CABG were recruited in Tabriz Madani Educational Center in a 15-month period of time. Patients were randomized into two groups of 60 receiving oral atorvastatin (40mg) plus oral vitamin C (2g/d operation day and 1g/d for five consequent days) for intervention group and oral atorvastatin (40mg) for control group. Occurrence of post CABG AF was compared between the two groups.

Results: There were 60 patients, 43 males and 17 females with a mean age of 61.0±11.5 (29-78) years, in the intervention group and sixty patients, 39 males and 21 females with a mean age of 60.5±11.3 (39-81) years, in the control group. The post CABG AF occurred in 6 cases (10%) in the interventional group and 15 patients (25%) in the controls (P=0.03, odds ratio=0.33, 95% confidence interval 0.12-0.93).

Conclusion: Based on our findings, combination prophylaxis against post CABG AF with oral atorvastatin plus vitamin C is significantly more effective than single oral atorvastatin.

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Effect of ascorbic acid on plasma cholesterol in humans in a long-term experiment.

During the period of a low vitamin C intake (approximately equal to 20 mg per day) ascorbic acid in a dose of 2 x 500 mg per day was administered to 82 men and women aged 50-75 years. 

A correlation of plasma cholesterol levels determined before and after a three months' administration of ascorbic acid showed the effect of vitamin C to be dependent on the starting concentration of plasma cholesterol: the higher the initial cholesterolemia, the greater the hypocholesterolemic effect of ascorbic acid. 

On restricting the experimental group to subjects with an initial cholesterolemia above 230 mg%, the effect of the same dose of ascorbic acid on cholesterolemia was followed in three-month periods for a further 9 months. 

In all these time intervals, ascorbic acid was found significantly to depress cholesterolemia and its effects persisted 6 weeks after termination of the experiment. 

The administration of 2 x 500 mg ascorbic acid daily during one year resulted in an abrupt increase of ascorbemia and a marked accumulation of ascorbic acid in the leucocytes. Six weeks following interruption of ascorbic acid intake, vitamin C concentration in the leucocytes significantly declined but still continued to be twice higher than in the control receiving no ascorbic acid supplement.

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Vitamin C treatment reduces elevated C-reactive protein

Plasma C-reactive protein (CRP) is an inflammatory biomarker that predicts cardiovascular disease. Lowering elevated CRP with statins has reduced the incidence of cardiovascular disease. 

We investigated whether vitamin C or E could reduce CRP. Healthy nonsmokers (N=396) were randomized to three groups, 1000 mg/day vitamin C, 800 IU/day vitamin E, or placebo, for 2 months. 

Median baseline CRP was low, 0.85 mg/L. No treatment effect was seen when all participants were included. 

However, a significant interaction was found, indicating that treatment effect depends on baseline CRP concentration. Among participants with CRP indicative of elevated cardiovascular risk (> or =1.0 mg/L), vitamin C reduced the median CRP by 25.3% vs placebo (p=0.02) (median reduction in the vitamin C group, 0.25 mg/L, 16.7%). These effects are similar to those of statins. 

The vitamin E effect was not significant. In summary, treatment with vitamin C but not vitamin E significantly reduced CRP among individuals with CRP > or =1.0 mg/L. Among the obese, 75% had CRP > or =1.0 mg/L. 

Research is needed to determine whether reducing this inflammatory biomarker with vitamin C could reduce diseases associated with obesity. 

But research on clinical benefits of antioxidants should limit participants to persons with elevations in the target biomarkers

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Nature's Perfect Statin

The hot selling statins, such as Lipitor® and Zocor®, lower cholesterol by blocking the coenzyme HMG-CoA reductase. The first statin drugs were released in 1987, and the manufacturing and promotion of these drugs has grown into a huge business; statins are the most widely prescribed class of drugs in history.

In 1985, scientists made an important discovery. Ascorbic acid (Vitamin C) is the human body's natural HMG-CoA reductase inhibitor.[*]  When vitamin C levels are low, the body compensates and manufactures more cholesterol; when levels are high, the vitamin inhibits HMG-CoA reductase, lowering cholesterol.

The problem with the artifical statin drugs has been identified. These drugs concurrently reduce circulating levels of ubiquinone, aka coenzyme Q10 (CoQ10) [*] and raise levels of the lipid Lp(a) [*] . A decline in CoQ10 -- attributed to artificial statins -- has been linked to muscle pain, muscle myopathy and congestive heart failure; [*] elevated Lp(a) is linked to the 70 percent higher probablity of heart attack or stroke [*].

Vitamin C is Nature's Perfect Statin.

At the proper (high) dosage vitamin C lowers Lp(a), and it promotes the natural production of ubiquinone (CoQ10).

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